Hi Saul, You seem to “blame” the victim for experiencing rumination by thinking without deciding. My understanding of rumination, both from published scientific journals and personal experience, is as follows: Ruminative responding in major depressive disorder (MDD) is defined as a recurrent, self-reflective, and uncontrollable focus on depressed mood and its causes and consequences (1-3). Higher levels of rumination have been found to predict both more severe depressive symptoms in depressed individuals (4) and the onset of depressive symptomatology in nondepressed people (5). Although ruminative responding is not considered a criterion symptom of depression in DSM-5 or ICD-10, measures of rumination nonetheless consistently (and often, perfectly, e.g., 6) differentiate depressed from never-depressed individuals. Indeed, theorists have posited that rumination is a central aspect of the phenomenology of MDD (7). If you wish to review the cited references, and I encourage you to do so, they are: References 1. Morrow J, Nolen‐Hoeksema S (1990): Effects of responses to depression on the remediation of depressive affect. Journal of Personality and Social Psychology. 58:519‐527. 2. Nolenhoeksema S (1991): RESPONSES TO DEPRESSION AND THEIR EFFECTS ON THE DURATION OF DEPRESSIVE EPISODES. Journal of Abnormal Psychology. 100:569‐582. 3. Whitmer AJ, Gotlib IH (in press): An Attentional Scope Model of Rumination. Psychological Bulletin. 4. Kuehner C, Weber I (1999): Responses to depression in unipolar depressed patients: an investigation of Nolen‐Hoeksema’s response styles theory. Psychological Medicine. 29:1323‐1333. 5. Nolen‐Hoeksema S, Wisco BE, Lyubomirsky S (2008): Rethinking Rumination. Perspectives on Psychological Science. 3:400‐424. 6. Hamilton JP, Furman DJ, Chang C, Thomason ME, Dennis E, Gotlib IH (2011): Default‐mode and task‐positive network activity in Major Depressive Disorder: Implications for adaptive and maladaptive rumination. Biological Psychiatry. 70:327‐733. 7. Lyubomirsky S, Tucker KL, Caldwell ND, Berg K (1999): Why ruminators are poor problem solvers: Clues from the phenomenology of dysphoric rumination. Journal of Personality and Social Psychology. 77:1041‐1060. Turning now to your “SNAP CLUB” game, am I correct in understanding the rules as: SNAP CLUB 1. Do anything you want, any time you want. 2. When you decide to do something, at the moment when you decide, SNAP YOUR FINGERS. As you may be aware, anhedonia is recognized as a hallmark symptom of depression. Anhedonia, the inability to experience pleasure, is included as a primary symptom in the diagnostic criteria for clinical depression in both the Diagnostic and Statistical Manual of Mental Disorders -Fourth Edition (DSM-IV; American Psychiatric Association, 2000) and the International Statistical Classification of Diseases and Related Health Problems (World Health Organization, 1992). Along with other symptoms used to clinically diagnose Major Depressive Disorder, are lack of interest, lack of enthusiasm, reduced attention span, reduced concentration, loss of energy, and excessive fatigue. While some people experiencing depressed mood may benefit from games and other diversionary activities, patients with Major Depressive Disorder or refractory depressive disorder may likely find the first rule overwhelming and implausible. Of course, I am referring to patients who suffer from severely treatment-resistant depressive disorder, as determined by one or more accepted assessment tools, i.e. the Antidepressant Treatment History Form (ATHF), the Thase and Rush Staging Model (TRSM), the European Staging Model (ESM), the Massachusetts General Hospital Staging Model (MGH-s), or the Maudsley Staging Model (MSM). If you doubt that treatment-resistant depression is somehow different from feeling depressed, I recommend you read some research on the subject. May I suggest: Treatment-resistant Depression: A Separate Disorder, Hans-Jürgen Möller, Florian Seemüller, Rebecca Schennach and Ramesh K. Gupta (2013), also Definitions and Predictors of Treatment-Resistant Depression, Daniel Souery and William Pitchot (2013). Lastly, you might find it informative to review: Toxic Effects of Depression on Brain Function: Impairment of Delayed Recall and the Cumulative Length of Depressive Disorder in a Large Sample of Depressed Outpatients, P. Gorwood, et al, Am J. Psych. 2008; 165:731-739. This article has been widely cited for its finding, gathered from over 8,000 patients, that “There is a current tendency to demean the significance of depressive symptoms as evidence of distress rather than illness. We would not seek to dispute the distress, but our data support the hypothesis that recurrent or prolonged depression has effects on the brain that make it a significant and disabling illness.” Also, “Atrophy of the hippocampus is one of the most consistent imaging findings in major depressive disorder. This has highlighted the potential role of physiological stress as a core mediating factor (often assuming a neurotoxic effect of cortisol on the hippocampus) and has emphasized the need for antidepressant treatments that might prevent or even reverse hippocampal atrophy.