Similarity of Dissociation and Voice-Hearing in DID and Schizophrenia

Kermit Cole

A study of 40 patients with schizophrenia diagnoses and 40 patients with dissociative identity disorder (DID) found that “neither phenomenological definitions of dissociation nor the current generation of dissociation instruments (which are uniformly phenomenological in nature) can distinguish between the dissociative phenomena of DID and what we suspect are just the dissociation-like phenomena of schizophrenia.” The study further found similarity between the experiences of hearing voices in both disorders. Results were published in the Journal of Trauma and Dissociation online on June 2, 2012.

Abstract →

Laddis, A. Dell, P.F; “Dissociation and Psychosis in Dissociative Identity Disorder and Schizophrenia.” Journal of Trauma and Dissociation, July, 2012; 13(4): 397-413

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Kermit Cole
Kermit Cole, MFT, founding editor of Mad in America, works in Santa Fe, New Mexico as a couples and family therapist. Inspired by Open Dialogue, he works as part of a team and consults with couples and families that have members identified as patients. His work in residential treatment — largely with severely traumatized and/or "psychotic" clients — led to an appreciation of the power and beauty of systemic philosophy and practice, as the alternative to the prevailing focus on individual pathology. A former film-maker, he has undergraduate and master's degrees in psychology from Harvard University, as well as an MFT degree from the Council for Relationships in Philadelphia. He is a doctoral candidate with the Taos Institute and the Free University of Brussels. You can reach him at [email protected]


  1. Basically, what it says is that dissociation in what is called DID, i.e. split-off parts of the self that take shape as different personalities, and dissociation in what is called schizophrenia, i.e. split-off parts of the self that take shape as voices, are very similar, and difficult to distinguish from each other with the currently available tools. Interestingly — though not surprisingly — the researchers seem to think that developing newer, more precise tools will make it easier to distinguish between what they presume is “real” dissociation, caused by trauma (as in “DID”), on the one hand, and “false” dissociation, caused by some brain defect (as in “schizophrenia”), on the other. It doesn’t occur to them that what they have studied might be two slightly different, although also closely related, types of very “real” dissociation, both caused by trauma. Cf. Ron Unger’s slides for a presentation, “When Trauma and Psychosis Mix”: Especially slides 29 – 33/34

  2. “I had conversations with top researchers, established names in both trauma and schizophrenia who at first denied the connections and then acknowledged them but added that pursuing that line of thinking was “a career ender.” To even raise the question marked you as a radical and, at the time, their areas of interest could not have been sustained by pursuing what seemed from their perspectives to be a side show.” _Kermit.

    Sadly what is routinely ignored in all cognitive constructs about “symptom” expression is the body & the nervous systems role in feedback to the brain. Research & knowledge which address this is ignored, presumably because it threatens our inter-subjective mode of maintaining a group dynamic equilibrium (homeostasis).

    People like Allan N Schore & Stephen Porges have a hard time getting a more holistic approach recognized.

    “Pathological dissociative detachment represents a bottom-line defensive state driven by fear-terror, in which the stressed individual copes by pervasively and diffusely disengaging attention “from both the outer and inner [italics added] worlds” (Allen et al., 1999, p. 164). I have suggested that the “inner world” is more so than cognitions, the realm of bodily processes, central components of emotional states (Schore, 1994).

    In line with the current shift from cold cognition to the primacy of bodily based affect, clinical research on dissociation is now focusing on “somatoform dissociation.” According to Nijenhuis (2000), somatoform dissociation is an outcome of early onset traumatization, expressed as a lack of integration of sensor motor experiences, reactions, and functions of the individual and his or her self representation.

    Thus, “dissociatively detached individuals are not only detached from the environment, but also from the self—their body, their own actions, and their sense of identity”.
    This observation describes impaired functions of the right hemisphere, the locus of the “emotional” or “corporeal self.” According to van der Kolk and colleagues (1996), “Dissociation refers to a compartmentalization of experience: Elements of a trauma are not integrated into a unitary whole or an integrated sense of self”

    In a number of works I have offered interdisciplinary evidence that indicates that the implicit self, equated with Freud’s System Ucs, is located in the right brain. The lower subcortical levels of the right brain (the deep unconscious) contain all the major motivational systems (including attachment, fear, sexuality, aggression, etc.) and generate the somatic autonomic expressions and arousal intensities of all emotional states. On the other hand, higher orbitofrontal-limbic levels of the right hemisphere generate a conscious emotional state that expresses the affective output of these motivational systems. This right lateralized hierarchical prefrontal system, the system Pcs. performs an essential adaptive motivational function—the relatively fluid switching of internal bodily based states (Bromberg’s self-states) in response to changes in the external environment that are nonconsciously appraised to be personally meaningful.

    On the other hand, pathological dissociation, an enduring outcome of early relational trauma, is manifest in a maladaptive highly defensive rigid, closed system, one that responds to even low levels of intersubjective stress with parasympathetic dorsal vagal parasympathetic heart rate hypo arousal and deceleration. This fragile unconscious system is susceptible to mind–body metabolic collapse, Janetian energy failure, and thereby a loss of energy-dependent synaptic connectivity within the right brain, expressed in a sudden implosion of the implicit self and a rupture of self-continuity.

    This collapse of the implicit self is signaled by the amplification of the parasympathetic affects of shame and disgust and by the cognitions of hopelessness and helplessness. Because the right hemisphere mediates the communication and regulation of emotional states, the rupture of intersubjectivity is accompanied by an instant dissipation of safety and trust.

    Dissociation thus reflects the inability of the right brain cortical– subcortical implicit self-system to recognize and process external stimuli (exteroceptive information coming from the relational environment) and on a moment-to-moment basis integrate them with internal stimuli
    (interoceptive information from the body, somatic markers, the “felt experience”).

    This failure of integration of the higher right hemisphere with the lower right brain induces an instant collapse of both subjectivity and intersubjectivity. Stressful affects, especially those associated with emotional pain, are thus not experienced in consciousness (Bromberg’s “not-me” self-states).

    The critical elements of implicit unconscious intersubjective communications embedded in affectively charged attachment transactions are more than mental contents; rather, they are interactively communicated and regulated and dysregulated psychobiological somatic processes that mediate shared conscious and unconscious emotional states. Recall Freud’s remark to Groddeck: “The unconscious is the proper mediator between the somatic and the mental, perhaps the long-sought ‘missing link’”.

    The essential biological purpose of intersubjective communications in all human interactions, including those embedded in the psychobiological core of the therapeutic alliance, is the regulation of right-brain/mind/body states.

    Andrade suggests that it is the affective link, conveyed through intersubjective “empathetic introjective reciprocal identification” (p. 694), that leads to both psychic and somatic change.” _Allan N Schore. “Affect Dysregulation & Disorders of the Self.”

    Core affect/emotion is suppressed for the sake of subjective harmony from around 18 months of age on-wards, and perhaps we all pay a price for an adult collusion of Descartes famous error?

    Is it the fear of infectious emotion that sees us prefer on “objectified” sense of self that allows this presumption of symptom expression, as separate parts like experience, and a continuum of body/brain/mind expression?